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The abundance of Lactobacillus in SLE patients has shown inconsistency across studies: some report an increase [16, 19], whereas others observe a reduction in newly diagnosed SLE patients [20]

Concurrently, the compromised intestinal barrier facilitates the translocation of microbial components, including LPS and flagellar proteins, along with secondary bile acids, into the systemic circulation, thereby triggering systemic inflammatory responses (103)

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