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Intestinal microbiome members induce immune tolerance and block pathogen colonization through the activation of the immune system and the direct and indirect actions of the microbiota

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Different agonists recruit different stromal interaction molecule proteins to support cytoplasmic Ca 2+ oscillations and gene expression

To validate these findings in a loss-of-function model, we performed ex vivo CRISPR/Cas9-mediated knockout of Gpr146 in SVF cells isolated from Cas9-expressing mice