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Qi S, Wang Q, Zhang J, Liu Q, Li C

Zhang L, Ma G, Yamawaki K, Ikoma Y, Matsumoto H, Yoshioka T, et al

Thus, TNBS promotes two independent mechanisms of injury, GSH depletion and ROS generation, both being required for the manifestation of mucosal injury as GSH limitation renders intestine susceptible to the TNBS-induced ROS overgeneration

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1209 These findings indicate that the combination of CRISPR-Cas9 gene editing technology with single-cell metabolomics and human organoids holds promise for accurately monitoring cellular metabolic changes in relevant diseases