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Intriguingly, depletion of the H3K27me3 mark before capecitabine treatment enhanced chemotolerance, whereas simultaneous treatment with both capecitabine and a KDM6 inhibitor (to increase global levels of H3K4me3) delayed tumour recurrence 50

10.1016/j.arr.2012.10.004 15

This indicates that an inflammatory environment has been induced in which the levels of potentially pathogenic cytokines are likely to cause, or contribute to the induction of neuronal damage or death ( It is now clear that there are also major changes in the immune system associated with some central disorders, of which Huntington's disease represents a good example

Ransy C, Vaz C, Lombs A, Bouillaud F