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Proteomics analysis revealed that miR-23a-3p inhibitors could prevent ACSL4 overexpression and reduce sorafenib-induced lipid peroxidation and cellular iron accumulation, suggesting that miR-23a-3p can affect ferroptosis by regulating ACSL4 to affect sorafenib resistance in hepatocellular carcinoma (23)

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It has been shown that exposure to microcystin-leucine-arginine and T-2 toxin suppresses the release of FSH in the pituitary gland, which, by reducing testosterone levels, decreases fertility (280, 281)

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And therein we clearly see the implications in infections, respiratory infections, in cancer, which is largely an immune system dysregulation