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Thus, site-specific oxygen radical production by doxorubicin may be responsible for doxorubicin-induced mitochondrial damage [99]

BPC-157 demonstrates promising cardiovascular effects through its influence on the nitric oxide system, which plays a central role in blood vessel dilation and blood pressure regulation

Mechanistically, mitochondrial dysfunction, as discussed above, is an increasingly recognized contributor, particularly in diabetic nephropathy, in which mitochondrial ROS production exceeds the local antioxidant capacity [13,34]

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Interleukin-6 signalling regulates vascular endothelial growth factor-C synthesis and lymphangiogenesis in human oral squamous cell carcinoma

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