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Studies have shown that ONOO can impair mitochondrial-ATP synthesis through inhibition of mitochondrial respiratory complexes, mitochondrial polarization, and detoxifying enzymes in addition to promoting mitophagy 65,66

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These oxidative insults activate microglia and astrocytes, triggering pro-inflammatory cytokine release (e.g., IL-1, TNF-) and propagating neuronal damage (Teixeira et al

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