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Further argument that BPC 157 had conducted a prompt counteraction in the face of L-NAME retinal ischemia may be the mentioned, quite general, counteracting potential of BPC 157 on the L-NAMEinduced damaging effects in different models and species (Stupnisek et al., 2015)

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Neurological Risk: Unbound free copper generates reactive oxygen species via Fenton chemistry

If not resolved, the continuous immune cell accumulation will be detrimental to an already damaged cardiac tissue [72]