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Abstract The intensive nutrient requirements needed to sustain T cell activation and proliferation, combined with competition for nutrients within the tumor microenvironment, raise the prospect that glucose availability may limit CAR-T cell function

doi: 10.1111/jfd.12610 147 MacKayW

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By binding directly to IGF-1 receptors on muscle cells, IGF-1 LR3 activates downstream anabolic pathways including PI3K/Akt and MAPK signalingpromoting protein synthesis, glucose uptake, cellular proliferation, and satellite cell activation

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