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Studies have indicated that cPLA2 is overactivated in ischemic brain tissue via N-methyl-D-aspartate (NMDA) receptor/Ca2+ and thrombin pathways, promoting the mobilization of AA (Xu et al., 2024

NAD kinase is essential for maintaining intracellular levels of reduced NADPH by catalyzing the conversion of NAD + to NADPH, one of the major intracellular reductants that plays a central role in the intracellular antioxidant defense system