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revealed that antibodies associated with the thromboinflammatory autoimmune disease antiphospholipid syndrome also triggered NET formation via glycolysis, the PPP and NADPH-dependent ROS production (Table 2) (Tambralli et al., 2024)

van Zandwijk N, Pavlakis N, Kao SC, Linton A, Boyer MJ, Clarke S, Huynh Y, Chrzanowska A, Fulham MJ, Bailey DL

However, some individuals may experience mild side effects, such as

Tissue-specific regulation of p53 by PKM2 is redox dependent and provides a therapeutic target for anthracycline-induced cardiotoxicity

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