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Iron overload not only promotes lipid peroxidation but also induces apoptosis and autophagy, further aggravating secondary brain injury (195)

Mitochondria-targeted catalase reduces abnormal APP processing, amyloid production and BACE1 in a mouse model of Alzheimers disease: Implications for neuroprotection and lifespan extension

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Persistent elevations in blood glucose initiate a series of metabolic and cellular disruptions that compromise vascular integrity as well as myocardial function

IL-1 treatment reduced SIRT1 expression and increased acetylated NF-B p65 (Lys310) without changing NF-B p65 protein levels (Fig