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This conversion can be inhibited by targeting mitochondrial ROS with antioxidants or selenium, further highlighting the importance of ROS in fibroblast-to-myofibroblast conversion [81, 82]

The IGF-I models created in this study and the validation experiments were mainly performed using L6 myoblasts, and it is unclear whether the dynamics of IGF-I signaling changes in the same manner with IGF-I concentration in different cell lines

10.1016/j.jns.2017.07.042 53

Although Lys360 and Lys384 were reported to be probable Sirt3-dependent deacetylation sites in murine Sirt3 knockout cells 24, 26 , their acetylation mimics did not exhibit significant decreases in IDH2 activity

They can reduce chronic inflammation that triggers cancer, and they can also reshape the inflammatory tumor environment itself

Overall, these data suggest that early acute inflammation is required for the correct recruitment of OPCs to the lesion site, but that chronic inflammation may impede remyelination (Figure 2)