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This review discusses and compares the mechanistic pathways involved in these two clinical presentations of this enzyme disorder

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[DOI] [PubMed] [Google Scholar] 74.Vulcano LA, Confalonieri O, Franci R, Tapia MO, Soraci AL

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Consequently, ubiquitin architectures with K29 linkages as foundation and K48 linkages as branches may survive in cells due to E3s/DUBs competition and serve as a productive signal for proteasomal degradation

Erratum: cloning and expression of a rat brain L-glutamate transporter