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[6] The concern with any unregulated IGF-1R agonist is precisely that it could stimulate the survival and proliferation of pre-neoplastic or neoplastic cells, which is why IGF-1R has been an oncology drug target for inhibition rather than activation

About this article Cite this article Smith, T.A., Kirkpatrick, D.R., Smith, S

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sativa resulted in a differential increase in antioxidant proteins, including OsPHGPX, especially in the early stages of a compatible interaction