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4 Role of piperine in targeting cancer hallmarks 4.1 Role of piperine in genomic instability Genomic instability is a fundamental hallmark of all cancer cells, enabling malignant cells to accumulate mutations and evade regulatory mechanisms

A previous study demonstrated that ischaemia/reperfusion injury (IRI)-induced immune cell infiltration of the cremaster muscle was mediated by ferroptosis, and Fer-1 directly inhibited leukocyte transmigration or created a milder proinflammatory microenvironment, which may have been caused by a decrease in DAMP release secondary to the decrease in ferroptosis 11, 60

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Functionalization with targeting ligands further enhances uptake by neurons or glia, enabling precise regional delivery

In addition, riboflavin-dependent enzymes have essential roles in pyridoxine activation, tryptophan-kynurenine pathway, and homocysteine metabolism

In patients with IBD, deficits in both the number and function of Tregs lead to immune dysregulation and exacerbation of inflammation [363]