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The results highlight nitrosative redox chemistry as a potentially physiologically relevant pathway for dsNNTTR formation and support a model in which controlled thiol modification, rather than generalized oxidative stress, may contribute to pathological TTR crosslinking in vivo

The cGAS-STING pathway activates transcription factor TFEB to stimulate lysosome biogenesis and pathogen clearance

203 found the systemic availability of the drug to be somewhat less and more variable (mean, 74%

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NF1 plexiform neurofibroma growth rate by volumetric MRI: relationship to age and body weight

Baldeiras I, Santana I, Proena MT et al (2008) Peripheral oxidative damage in mild cognitive impairment and mild Alzheimers disease