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LL-37 can also bind bacterial lipopolysaccharide (LPS) and block its interaction with CD14/TLR4, reducing endotoxin-triggered TNF release and neutrophil apoptosis [2]

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On the contrary, pitavastatin and, to a lesser extent, atorvastatin and simvastatin would increase ABCA1 expression in hepatocytes (Maejima et al., 2011)

Abstract Hyperhomocysteinemia is an independent risk factor for atherosclerotic diseases

CBS overdosage is necessary and sufficient to induce cognitive phenotypes in mouse models of Down syndrome and interacts genetically with Dyrk1a