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Increase in ascorbate-glutathione cycle enzymes activities, CAT, ascorbate-glutathione content, SOD, and ascorbate-glutathione redox clearly evident that CAT, ascorbate-glutathione cycle and SOD played a significant activity in ROS detoxification of tolerant A

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Further research confirms that ACSL4 suppression in early ischemic stages is time-dependent and likely driven by increased HIF-1, which binds to the ACSL4 promoter and suppresses its expression (137)