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doi: 10.3389/fimmu.2019.00423 54 MuraoAAzizMWangHBrennerMWangP

J Pineal Res (2012) 52(3):28290

Yamamoto, M., Hibi, H., Hirata, Y., Miyake, K

Cap analysis of gene expression (CAGE) sequencing reveals alterations of the transcript signatures in acute monocytic leukemia cells by fatty acid oxidation inhibition

The excess mortality reported in patients with CKD and high ALP levels has been controversially attributed to potential cardiovascular adverse effects as a result of very severe lack of control over bone-mineral metabolism.2,3,13,14 Several pathogenic mechanisms have been proposed to explain this association, including increased vascular calcification via pyrophosphate hydrolysis in the arterial wall,15 systemic inflammation16,17 and vitamin D deficiency.18 However, the absence of a significant association between serum levels of the bone-derived isoenzyme of ALP (bone-specific ALP) and mortality in patients with CKD19 raises serious doubts about the hypothesis on a pathogenic link with bone-mineral metabolism

Development and characterization of minoxidil-loaded liposomal system for delivery to pilosebaceous units