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Copper supplementation in both animal models and humans (e.g., patients with SCO2 mutations) can restore CCO activity, reverse hypertrophy and fibrosis, and significantly improve cardiac function [51, 52]

This data suggests once again a synergistic role between FOXO isoforms and their contribution to hepatic glucose production and insulin resistance

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In senescent cells, which are cells that have ceased to divide and accumulate in tissues, this interaction helps them resist apoptosis

Motzer RJ, Escudier B, McDermott DF, George S, Hammers HJ, Srinivas S, Tykodi SS, Sosman JA, Procopio G, Plimack ER, et al