Mol Cell Biol 24:87788789
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Conversely, others and our laboratory have demonstrated that NO can favor GSH synthesis via the NEFL2-mediated transcription of GCL ( The role of GSH as NO scavenger is also highlighted by To investigate the importance of correct GSH homeostasis for the neurotrophic capacity of NO, 2 -Tyr) protein accumulation ( Figure 1 )
In vivo rodent longevity models have examined the compound's influence on tissue-specific telomere length dynamics across serial sampling timepoints
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