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(23), which analysed the copy number of mtDNA in the blood, revealed that changes in the copy number of mtDNA elicited mitochondrial dysfunction, indicating that in the future, the quantification of the copy number of mtDNA may provide an opportunity to targeted treatment for related diseases

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de Moraes MeineBFranceschiTSBonaNPSpohrLPedraNSSpanevelloRMet alChemical hypermethioninemia in young mice: oxidative damage and reduction of antioxidant enzyme activity in brain, kidney, and liver

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