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showed that intracellular glutamate accumulation following cyst(e)ine depletion or system x C - inhibition is a key contributor to the induction of ferroptosis, and glutamate toxicity can be alleviated by converting excess glutamate to -glutamyl-di- or tripeptides via promiscuous enzymatic activities of glutamate-cysteine ligase catalytic subunit (GCLC) 12

This means that thyroid cells need to maintain their level of oxidative stress within a certain range, rather than uniformly attempting to lower it by producing more anti-ROS enzymes and molecules like glutathione

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Xu C, Wang F, Su C et al (2023) Restoration of aquaporin-4 polarization in the spinal glymphatic system by metformin in rats with painful diabetic neuropathy