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Silicon-induced lung injury may result from multiple interacting pathological mechanisms, including the damage caused to macrophages by the phagocytosis of SiO 2 , the reactive oxygen species (ROS) production, mitophagy and apoptosis (Zhang et al., 1997), stem cell therapy (Zhang et al., 2018) and even lung transplantation ( Hydrogen (H 2 ), as a gaseous therapeutic agent, can directly penetrate the lungs without requiring systemic absorption, thereby exerting its effects locally