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Therefore, mitochondrial metabolic rewiring not only sustains cellular homeostasis but also functions as a pivotal node in immunometabolic cross-talk, contributing to the pathogenesis of chronic inflammatory disorders (such as RA)

These cytokines disrupt the production of key ECM proteins, including collagen type II and aggrecan, while promoting the synthesis of matrix metalloproteinases (MMPs), which leads to ECM degradation and further progression of OA [2]

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To evaluate the effect of BSA-AGE on BMDCs maturation, BMDCs were stimulated with 10 g/mL of LPS or 100 g/mL of BSA-AGE for 18 h at 37 C

The relationship between this response and both glutathione homeostasis and overall antioxidant capacity is not yet clear, and additional research is planned to investigate this further