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Rallabhandi, P

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With a deeper understanding of ferroptosis during the occurrence and progression of cerebral I/R injury, new therapies to treat ischemic nerve damage by interfering with the molecular mechanism of ferroptosis are expected to be realized to some extent

and experimental data are consistent with a role for ergothioneine in erythroid proliferation and differentiation (Reference Nakamura, Sugiura and Kobayashi33)

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NMN directly enters the NAD+ production pathway