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Although cellular senescent pathways have been implicated in IPF pathology and in fibroblasts isolated from IPF patients 59,60 , other studies report TGF-induced FMT alterations in fibroblast monocultures 7,8,9

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Eotaxin is specifically cleaved by hookworm metalloproteases preventing its action in vitro and in vivo

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6 These metabolic disturbances primarily induce endoplasmic reticulum (ER) stress, oxidative deficit, and mitochondrial dysfunction, leading to -cell damage and dedifferentiation