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A further study examined whether SAMe could delay neuropathology in 3Tg-AD mice, which harbor mutant genes for human APP, PS-1, and tau, here showing that SAMe supplementation in the diet did, in fact, reduce extracellular A deposits [196]

Cancer Res 80(22):50985108

Therefore, to regulate the metabolism of iron without affecting these important roles and affect ferroptosis and then treat tumors, a comprehensive understanding of its molecular mechanisms is required

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The study found that after IDH1 mutation, it will catalyze the massive conversion of 2-KG to tumor metabolite 2-HG, while consuming reduced NADPH