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This suggests that the proliferation and functionality of immune cells after the recall response was similar in all treatment groups

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In response to lung injury, macrophages activated by LPS and IFN undergo a transition to the pro-inflammatory M1 phenotype and begin to secrete pro-inflammatory cytokines and chemokines, resulting in enhanced chemotaxis of monocytes and neutrophils (117, 118)

The variables become hopelessly confounded

doi: 10.1016/j.semcdb.2019.04.016