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As a side note, AUC is the best way to measure bioavailability because it takes into account the entire response over time, whereas Cmax, which is used by some researchers, measures only one point in time and is, therefore, less reliable

This overlap opens the discussion about the possibility of common pathogenic pathway.[2] References: 1

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As MMPs depend on zinc for their activation, zinc exerts precise control over inflammatory responses by inhibiting MMP-1-mediated lymphocyte infiltration and preventing MMP-9-induced basement membrane disruption (22, 23)

Mitochondrial injury in AP is accompanied by decreased m, elevated ROS levels, activation of the NLRP3 inflammasome, and pyroptosis, ultimately leading to PAC necrosis (73)