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Thus, since iron levels in macrophages may drive cholesterol efflux, targeting macrophages environment manipulating iron levels and/or iron metabolism-related molecules could interfere with the generation of foam cells and the development of atherosclerosis

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In order to test this possibility, we engineered a murinized version of the anti-IGF-1R mAb, L2-C (L2-Cmu), in order to reduce effector function and enable chronic administration in mice