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These are due to its ability to increase glutathione levels, scavenge free radicals and reduce oxidative stress

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De Vallire C, Cosin-Roger J, Simmen S, Atrott K, Melhem H, Zeitz J, et al

320,321,322 Mechanistically, PRIMA-1 functions through its degradation product, the methylene quinuclidinone (MQ)

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In animal models, it has been reported that the activation of apoptosis in type II AEC promotes a fibrotic phenotype, for example, apoptotic cells may release cytokines and growth factors that recruit and activate fibroblasts, prompting them to secrete ECM, whereas inhibiting apoptosis helps to alleviate pulmonary fibrosis (102104)