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Hepatocyte-specific deletion of IRE1 abrogates IRE1-XBP1 activation and disrupts fatty acid oxidation and ketogenesis, suggesting the failure of the adaptive metabolic shift to fasting

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The N-terminal YGGF motif of opioid peptides occupies a conserved activation chamber at the bottom of the orthosteric binding pocket, which is crucial for receptor activation

Similar reciprocity was observed at the redox-sensitive cysteine of GAPDH Cys152 ( Fig