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Notably, the NRF2 inhibitor Brusatol provides a compelling mechanistic example: it triggers ferroptosis in HNSCC models (including ESCC and OSCC) by promoting NRF2 degradation, which subsequently represses the expression of key genes like GCLC, thereby depleting GSH, enlarging the labile iron pool, and driving the accumulation of lethal lipid ROS (Zhu et al., 2023

PMID 543550.

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Defects in this gene result in familial adenomatous polyposis (FAP), an autosomal dominant precancerous condition that often progresses to malignancy

C.ZhaoQ.et al (2016)

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