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In addition, albumin nanoparticles can bypass the hENT1-dependent uptake pathway due to their natural targeting (binding to the tumor microenvironment through SPARC protein) and unique transmembrane mechanisms (such as gp60 receptor mediated transport), directly increasing the concentration of gemcitabine in resistant tumor cells, and have shown advantages in overcoming hENT1-deficient resistance in pancreatic cancer and bladder cancer models (Bartkowski et al., 2024

Journal of molecular biology 310 , 895906, doi: 10.1006/jmbi.2001.4790 (2001)

Demarquoy J, Le Borgne F (2015) Crosstalk between mitochondria and peroxisomes

4d, e and Supplementary Figure S1d-e, CQ inhibited the maturation of autophagy, resulting in a predominance of autophagosomes (yellow) in cells, whereas in Rap-treated or GPx1-shRNA-expressing cells, only some of the LC3-positive puncta were yellow

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