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It has been investigated for its involvement in angiogenic signaling , where it is referenced in the literature as a modulator of vascular growth factor pathways

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When you shift the goal from eliminating stress to supporting your response to it, everything becomes more manageable and more sustainable

ADAM proteases mediate shedding of the extracellular domain of c-MET, generating a soluble N-terminal fragment that acts as a decoy to antagonize c-MET dimerization and signaling [5]

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