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Furthermore, iron chelators such as minocycline have shown the ability to inhibit the activation and proliferation of microglia and matrix metalloproteinases

This is exciting as it could allow for more general control of protein activity by CPS, given that control is linked to transcriptional activation, which could theoretically control any gene, rather than linked to interrupting a specific target protein by post-translational control

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Human microglia express roughly 50% fewer nanoparticle-targeting receptors, such as triggering receptors expressed on Myeloid cells 2 (TREM2), than rodent models, reducing cellular uptake [221]