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Generally, MEC-1 cells were less sensitive to CB-839 than were wild-type HG-3 cells, potentially because of the MEC-1 cells complex karyotype, including del(17p)

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demonstrated that suppressing the expression of SLC31A1, a key mediator of cellular Cu uptake in eukaryotic cells [Citation10], rescued renal function, improved mitochondrial activity, and inhibited apoptosis in AKI mice [Citation11]

Since many cell types are able to export GSH, the present results suggest that this could be a fundamental self-regulation of cell volume, giving the cells a self-control on their adhesion proteins

[20] It is important to note that doses higher than 600 mg/day intravenously have not been shown to confer additional clinical benefit, but they may increase side effects

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