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Inducing disulfidptosis in tumors: potential pathways and significance

Niacinamide supplementation helps maintain NAD+ levels, which supports mitochondrial function, DNA repair, and overall metabolic efficiency

The biological context in which a patient is exposed to PBDE from within the environment and PDBE is processed by the placenta and its direct metabolites and other factors (growth, cytokines/chemokines, etc

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Human umbilical cord mesenchymal stem cells alleviate chemotherapy-induced premature ovarian insufficiency mouse model by suppressing ferritinophagy-mediated ferroptosis in granulosa cells

Oxidation-sensitive linkers leverage elevated reactive oxygen species (ROS) levels in tumor and inflammatory sites, utilizing boronate esters, thioethers, or selenoethers that cleave under oxidative conditions to release drugs