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Keywords Reactive nitrogen species, reactive oxygen species, endoplasmic reticulum stress, unfolded protein response, drug-induced liver injury Introduction Drug-induced liver injury (DILI) plays an essential role both in the clinic and in drug development [1]

Ploemen JH, van Ommen B, Bogaards JJ, van Bladeren PJ

Current therapeutic approaches for FRDA, including HDAC inhibitors and gene replacement therapy, are under development, with promising results reported in preclinical models, but their efficacy in rescuing FRDA phenotypes remains to be fully determined

Although exclusion criteria included history of hypersensitivity reactions to NAC, we did not specifically exclude patients with asthma

Taken together, the prioritized Glu/Gln pathway in IPH+ plaque seems to implicate shifts in macrophage metabolism

This stability is critical for cell-based and in vivo research applications, where native peptides are rapidly degraded by intracellular proteases