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Role of mitochondrial dysfunction and dysregulation of Ca(2+) homeostasis in the pathophysiology of insulin resistance and type 2 diabetes

doi: 10.3390/ijms23105354 102 MaoZ

patients with mutations that lead to no MDR3 protein expression are not responsive to UDCA therapy [13]

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Mycobacterium lepraes evolution and environmental adaptation

This framework leverages the evolving multifunctional roles of neutrophils in HIRI, from a 'pro-inflammatory (N1)' to an 'anti-inflammatory/reparative (N2)' phenotype, to design alternative therapies for pro-inflammatory diseases