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Thus, we aimed at hypothesizing and providing some possible mechanisms linking oxidative stress to autoimmunity aspects and its implications for HT, as well as adjuvant therapeutic proposals to mitigate the deleterious effects

10.1016/j.fct.2022.113091 70 JuckerM.WalkerL

These findings demonstrate that TSA exerts neuroprotective effects by targeting the inhibition of the TLR4/NF-B signaling cascade, alleviating neuroinflammation following cerebral ischemia-reperfusion injury (137)

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It is clear from work in the 1970s and 1980s that the coefficient can be affected by several methodological factors, as discussed