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Future research is necessary to scrutinize cellular and molecular mechanisms of action of AR in cancer cells and the tumour microenvironment, to develop selective modulators of AR activity, and to identify patients with non-prostatic cancer who might benefit from targeting this pathway

doi: 10.3390/cancers13112716 14 JiangHYeYWangMSunXSunTChenYet al

Signal peptides in the proteins coded for in the C

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310 Pro-inflammatory activation further disrupts the TCA cycle, leading to substrate accumulation (e.g., citrate, succinate, and aconitate)

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