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Reduced FAK-STAT3 signaling contributes to ER stress-induced mitochondrial dysfunction and death in endothelial cells

doi: 10.1007/s40588-020-00140-w Summary Keywords COVID-19, necrosis, mechanisms, biomarkers, clinical treatment Citation Sun C, Han Y, Zhang R, Liu S, Wang J, Zhang Y, Chen X, Jiang C, Wang J, Fan X and Wang J (2022) Regulated necrosis in COVID-19: A double-edged sword

The ubiquitin hydrolase (Uch-L1) has been proposed to increase cellular levels of mono-ubiquitin and hence to increase rates of protein turnover by the UPS