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Following its translocation from the endoplasmic reticulum to perinuclear vesicles, activated STING enlists and activates TANK-binding kinase 1 (TBK1) [218]

Although Circular ASAP2 mediates DN, nothing is known about its biological mechanism and function

and the general neglect of the potential cell-specific functions of AhR signaling across different liver cell populations (e.g., hepatocytes, stellate cells)

The major influencing factors are the AATs

QUE also reversed DOX resistance in the MCF-7/DOX-resistant BC cell line by down-regulating the SNAI2, PLAU, and CSF1 genes (49)

The difference of cystine uptake between tumor cells and T cells indicates that an increase in cystine levels in the TME can promote not only the growth of T cells but also the disulfidptosis of tumor cells [72]