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doi: 10.14348/molcells.2023.0028 120 TakagiY.NakamuraT.NishiyamaA.NozakiK.TanakaT.HashimotoN.et al

Even though G6PD deficiency was inferred from the SNP detected in the GWAS data, metabolomics analyses confirmed that RBC from the donors bearing those SNP were indeed characterized by an ablation of the PPP and complete depletion of the total glutathione pool, including GSH but also its oxidized form, GSH-adducts (Figure 7A)

Several receptor antagonists, including YIL781, TAK-779, and AMD3100, were used to parse the involvement of specific receptor pathways, particularly the CXCR4 receptor, in mediating the observed effects [2]