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Reinke, H

Dysregulation of CTR1 and ATP7A/B, whether through altered expression (e.g., CTR1 downregulation in drug-resistant tumors) or genetic mutations, disrupts copper homeostasis and impairs drug accumulation

Furthermore, clinical data indicated that anti-angiogenic treatments are not always efficient in normalizing the tumor vasculature and could lead to therapy resistance due to the presence of compensatory angiogenic pathways (151)

Induction of apoptosis by the combination treatment of E 2 plus BSO was evidenced by changes in Bcl-2 and Bax expression

Modulates over 4,000 human genes and stimulates collagen synthesis by up to 70%

Concurrently, there were indications that under DSIP, the body still responded to environmental physical stimuli