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who suggested that acetaminophen ingestion depletes intracellular GSH stores, which then causes loss of feedback inhibition of -glutamylcysteine synthetase activity (Figures 3a and 3b).This increases production of -glutamylcysteine, which is partially converted to pyroglutamic acid

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Most striking, IAPP toxic oligomers appear to disrupt membranes of the secretory pathway, and then when adjacent to mitochondria, disrupt mitochondrial membranes ( Figure 4 illustrates schematically the process by which Akkermansia spp protect the host from type II diabetes

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Intern Med , 2003, 42(6): 483-489

Reactive nitrogen species mediate oxidative stress and astrogliosis provoked by in vivo administration of phytanic acid in cerebellum of adolescent rats: a potential contributing pathomechanism of cerebellar injury in peroxisomal disorders