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Western blot analysis of the protein levels of SLC7A11 showed an increased amount of SLC7A11 (Figure 5B)

Since Bcl-xl is a key mediator of mitochondrial function and cellular apoptosis that has been shown to bind to VDAC1 and increase the substrate permeability, its suggested that mTOR could control mitochondrial metabolism in a Bcl-xl-VDAC1 dependent manner (106)

No one thought I would return to the gym so quickly. Mark admits there were tough days in physical therapy

human saphenous vein endothelial cells [127]

152 Defects in damage recognition or apoptotic signaling may lead to relative resistance

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